Lupus (Systemic Lupus Erythematosus or SLE)

The lupus blood test can provide an accurate reading of the presence of antinuclear antibodies. 97% of people with lupus will test positive for the ANA that connects to the cell's nucleus or command center and damages or destroys the cells.  

Learn about your health today and order your labs directly from Ulta Lab Tests to screen, diagnose, and monitor for Lupus (Systemic Lupus Erythematosus) (SLE). 

Below the list of tests is a guide that explains and answers your questions on what you need to know about SLE tests, along with information on Systemic Lupus Erythematosus, signs, symptoms, and diagnosis.

 


Name Matches

Antinuclear antibodies are associated with rheumatic diseases including Systemic Lupus Erythematous (SLE), mixed connective tissue disease, Sjogren's syndrome, scleroderma, polymyositis, CREST syndrome, and neurologic SLE. 

Reflex Information: If ANA Screen, IFA is positive, then ANA Titer and Pattern will be performed at an additional charge.



ANAlyzeR™ ANA, IFA with Reflex Titer/Pattern, Systemic Autoimmune Panel 1

Includes

  • ANA Screen,IFA, with Reflex to Titer and Pattern
  • DNA (ds) Antibody, Crithidia IFA with Reflex to Titer
  • Chromatin (Nucleosomal) Antibody
  • Sm Antibody
  • Sm/RNP Antibody
  • RNP Antibody
  • Sjogren's Antibodies (SS-A, SS-B)
  • Scleroderma Antibody (Scl-70)
  • Jo-1 Antibody
  • Centromere B Antibody
  • Complement Component C3c and C4c
  • Cardiolipin Antibodies (IgA, IgG, IgM)
  • Beta-2-Glycoprotein I Antibodies (IgG, IgA, IgM)
  • Rheumatoid Factor (IgA, IgG, IgM)
  • Cyclic Citrullinated Peptide (CCP) Antibody (IgG)
  • 14.3.3 eta Protein
  • Thyroid Peroxidase Antibodies (TPO)

 

  • If ANA Screen, IFA is positive, then ANA Titer and Pattern will be performed at an additional charge (CPT code(s): 86039).
  • If the DNA (ds) Antibody Screen is positive, then DNA (ds) Antibody Titer will be performed at an additional charge (CPT code(s): 86256).

 

Alternative Name(s)

Expanded ANA Antibodies,Systemic Autoimmune Disorder,ANA and Expanded AI Testing,ANA and Systemic Autoimmunity,Comprehensive AI Testing,Early Systemic Autoimmune Disease,Autoimmune Disorders


Testing for anti-neutrophil cytoplasmic antibodies (P-ANCA and/or C-ANCA and/or atypical P-ANCA) has been found to be useful in establishing the diagnosis of suspected vascular diseases, inflammatory bowel disease, as well as other autoimmune diseases.

Testing for anti-neutrophil cytoplasmic antibodies (P-ANCA and/or C-ANCA) has been found to be useful in establishing the diagnosis of suspected vascular diseases (e.g., crescentic glomerulonephritis, microscopic polyarteritis and Churg-Strauss syndrome), bowel disease (Crohn's Disease, ulcerative colitis, primary sclerosing cholangitis, and autoimmune hepatitis) as well as with other autoimmune diseases (drug-induced lupus, SLE, Felty's syndrome). ANCA has classically been divided into C-ANCA and P-ANCA depending on the immunofluorescent pattern observed. More recently the specific antigens responsible for these patterns have been described and isolated. The antigen that gives the C-ANCA pattern is proteinase-3 (PR-3). Multiple antigens are responsible for P-ANCA pattern, the principle antigen being myeloperoxidase (MPO). Patients with vascular diseases will generally have either a C-ANCA pattern or P-ANCA pattern, and give positive results in specific tests for PR-3 or MPO. Patients with bowel disease have been shown to have antibodies that give a P-ANCA or C-ANCA pattern. These antibodies however, may not be directed towards MPO. Patients with drug induced lupus, etc., often present with a P-ANCA pattern that is associated with antibodies against MPO.

Beta-2-microglobulin normally passes through the glomerulus into the proximal tubule where much of it is reabsorbed. Serum levels are therefore an index of glomerular function. When impaired, serum levels rise in inverse ratio to glomerular filtration rate. Increased amounts of beta-2-microglobulin are excreted in several renal disorders, e.g., Balkan nephropathy, heavy metal poisoning and renal tubular disease due to therapeutic agents. Serial levels of beta-2-microglobulin in serum and urine are used to evaluate transplant viability and anticipate rejection. Following a successful graft, serum levels decline toward normal. Increasing serum levels provide an early sign of rejection. Elevated levels are also noted in lymphproliferative disorders, neoplasms (malignant and benign), inflammatory disease, and autoimmune diseases such as systemic lupus erythematosus (SLE) and Sjögren's disease

Increased CRP levels are found in inflammatory conditions including: bacterial infection, rheumatic fever, active arthritis, myocardial infarction, malignancies and in the post-operative state. This test cannot detect the relatively small elevations of CRP that are associated with increased cardiovascular risk.

C3a desArg is a cleavage product of C3 complement component activation. Elevated levels of C3a have been reported in patients with acute lyme disease, acute pancreatitis, systemic lupus erythematosus, and adult respiratory distress syndrome.


Cardiolipin Antibodies (IgA, IgG, IgM)

  • Cardiolipin Antibody (IgA) 
  • Cardiolipin Antibody (IgG) 
  • Cardiolipin Antibody (IgM) 

Cardiolipin antibodies (CA) are seen in a subgroup of patients with autoimmune disorders, particularly systemic lupus erythematosus (SLE), who are at risk for vascular thrombosis, thrombocytopenia, cerebral infarct and/or recurrent spontaneous abortion. Elevations of CA associated with increased risk have also been seen in idiopathic thrombocytopenic purpura, rheumatoid and psoriatic arthritis, and primary Sjögren's syndrome.


Cardiolipin Antibodies (IgG, IgM)

  • Cardiolipin Antibody (IgG) 
  • Cardiolipin Antibody (IgM) 

Cardiolipin antibodies (CA) are seen in a subgroup of patients with autoimmune disorders, particularly systemic lupus erythematosus (SLE), who are at risk for vascular thrombosis, thrombocytopenia, cerebral infarct and/or recurrent spontaneous abortion. Elevations of CA associated with increased risk have also been seen in idiopathic thrombocytopenic purpura, rheumatoid and psoriatic arthritis, and primary Sjögren's syndrome.


Cardiolipin antibodies (CA) are seen in a subgroup of patients with autoimmune disorders, particularly Systemic Lupus Erythematosus (SLE), who are at risk for vascular thrombosis, thrombocytopenia, cerebral infarct and/or recurrent spontaneous abortion. Elevations of CA associated with increased risk have also been seen in idiopathic thrombocytopenic purpura, rheumatoid and psoriatic arthritis, and primary Sjögren's syndrome.

Cardiolipin antibodies (CA) are seen in a subgroup of patients with autoimmune disorders, particularly Systemic Lupus Erythematosus (SLE), who are at risk for vascular thrombosis, thrombocytopenia, cerebral infarct and/or recurrent spontaneous abortion. Elevations of CA associated with increased risk have also been seen in idiopathic thrombocytopenic purpura, rheumatoid and psoriatic arthritis, and primary Sjögren's syndrome.

Cardiolipin antibodies (CA) are seen in a subgroup of patients with autoimmune disorders, particularly Systemic Lupus Erythematosus (SLE), who are at risk for vascular thrombosis, thrombocytopenia, cerebral infarct and/or recurrent spontaneous abortion. Elevations of CA associated with increased risk have also been seen in idiopathic thrombocytopenic purpura, rheumatoid and psoriatic arthritis and primary Sjögren's syndrome.

Decreased C3 may be associated with acute glomerulonephritis, membranoproliferative glomerulonephritis, immune complex disease, active systemic lupus erythematosis, and generalized autoimmune processes.

Decreased C3 and C4 levels may be associated with acute glomerulonephritis, membranoproliferative glomerulonephritis, immune complex disease, active systemic lupus erythematosis, cryoglobulinemia, congenital C4 deficiency and generalized autoimmune disease

Decreased C4 level is associated with acute systemic lupus erythematosis, glomerulonephritis, immune complex disease, cryoglobulinemia, congenital C4 deficiency and generalized autoimmune disease

CH50 is a screening test for total complement activity. Levels of complement may be depressed in genetic deficiency, liver disease, chronic glomerulonephritis, rheumatoid arthritis, hemolytic anemias, graft rejection, systemic lupus erythematosis, acute glomerulonephritis, subacute bacterial endocarditis and cryoglobulinemia. Elevated complement may be found in acute inflammatory conditions, leukemia, Hodgkin's Disease, sarcoma, and Behcet's Disease.

A Complete Blood Count (CBC) Panel is used as a screening test for various disease states including anemia, leukemia and inflammatory processes.

A CBC blood test includes the following biomarkers: WBC, RBC, Hemoglobin, Hematocrit, MCV, MCH, MCHC, RDW, Platelet count, Neutrophils, Lymphs, Monocytes, Eos, Basos, Neutrophils (Absolute), Lymphs (Absolute), Monocytes(Absolute), Eos (Absolute), Basos (Absolute), Immature Granulocytes, Immature Grans (Abs)


See individual tests

These studies contribute to the diagnosis of autoimmune blistering diseases, dermatitis herpetiformis, and IgA vasculitis. Important in the evaluation of other autoimmune and inflammatory skin diseases including non-IgA vasculitides and interface and lichenoid dermatitis/mucositis such as connective tissue disorders, lichen planus, etc. The panel includes the C5b-9 antibody to better distinguish between some forms of Lupus and Dermatomyocitis. Its presence or absence is also critical in the evaluation of other types of connective tissue diseases and many vasculitides.

Direct Antiglobulin Test (DAT) with Reflex to Anti C3 and Anti IgG

IMPORTANT - NOTE THIS IS A REFLEX TEST AND AN ADDITIONAL CHARGE OF $64 WILL OCCUR IF THE QUEST RUNS THE REFLEX TEST.

If DAT (Coombs, Direct) is positive, Anti C3d and Anti IgG will be performed at an additional charge of $64.00

Reference Range(s)

Negative

Clinical Significance

The DAT (Direct Coomb's test) is positive if red cells have been coated, in vivo, with immunoglobulin, complement, or both. A positive result can occur in immune-mediated red cell destruction, autoimmune hemolytic anemia, a transfusion reaction or in patients receiving certain drugs.

 


dsDNA Antibody is detected in patients with active systemic lupus erythematosus (SLE) and approximately 20% of patients with Mixed Connective Tissue Disease.

In conjunction with clinical findings and other laboratory tests, the high avidity dsDNA FARRZYME can be used as an aid to the diagnosis of systematic lupus erythematosus (SLE)


Preferred test in patients with Lupus anticoagulant, on heparin or direct thrombin inhibitors, or on Factor VIII concentrates.


Lupus refers to a chronic inflammatory autoimmune disorder.  This means it impacts the immune system by causing an excessive immune response against the body’s own tissues. In general, the immune system is designed to protect the body from external toxins and/or infections, but lupus causes the system to turn on its own body.  This causes the immune system to go after the blood vessels, joints, skin, and internal organs such as the lungs, brain, kidneys, and heart. It’s important to note there are several types of lupus; however, the most common is systemic lupus erythematosus (or SLE), and it affects a significant portion of the human body. 

Lupus and its symptoms can occur at any age but are most common in women of child-bearing age.  It is also far more common among women than men by a ratio of 10:1. In terms of a person’s ancestry, those coming from African, Hispanic, Asian, and Native American descent are most likely to be diagnosed with lupus.  With systemic lupus erythematosus, people between the ages of 15 and 44 are most prone to being diagnosed with it, but the condition is also seen in newborns and older adults. 

The root cause of lupus is still to be determined.  However, medical professionals and researchers state it likely involves a genetic component along with a personal trigger, which can vary from person to person, such as exposure to sunlight, viral infections, and/or specific prescription medications.  Lupus is also known to co-exist with other autoimmune disorders such as Hashimoto thyroiditis, hemolytic anemia, Sjögren syndrome, and idiopathic thrombocytopenic purpura (or ITP).  

 

There are multiple types of lupus, and here are the most common ones:

  • Systemic lupus erythematosus (or SLE) – This is the most common type of lupus and is noted for beginning with a subtle selection of signs/symptoms.  The Centers for Disease Control and Prevention states that approximately 16,000 new cases of SLE pop up in America each year, while 1.5 million Americans live with the condition. From the 1.5 million Americas that are already living with lupus, 90% of these cases involve women of child-bearing age.  SLE is known for targeting various parts of the human body at a systemic level. 
  • Discoid lupus – This is a type of lupus that is known for the development of chronic skin rashes. This rash tends to spread along the face and/or scalp.  Discoid lupus carries a 15-20% chance of turning into SLE.  
  • Subacute cutaneous lupus – This is a type of lupus that brings along with it skin lesions that are spread across multiple parts of the body subject to sun exposure. 
  • Drug-induced lupus – This is a type of lupus that is triggered through the use of specific drugs, which can include high blood pressure, anti-seizure, and anti-thyroid medications.  The particular medications include hydralazine (treatment for hypertension), procainamide (treatment of abnormal heartbeat patterns), and antibiotic isoniazid (treatment for tuberculosis).  With this type of lupus, the patient develops various symptoms a few months after the drug has established itself in the system.  However, these symptoms can go away once the medication has been stopped. 
  • Neonatal Lupus – This is a type of less common lupus that affects newborn babies leading to a set of symptoms such as liver problems, skin rash, and low blood cell counts.  These symptoms take a few months to go away, and, in many cases, a newborn has lupus because their mother already has SLE or Sjögren syndrome.  However, a newborn baby can be diagnosed with neonatal lupus even if their mother hasn’t been diagnosed with any form of lupus.  Other autoantibodies can simply trigger it in a mother’s bloodstream, such as anti-SSB/anti-SSA, which impact the fetus.  This is why women have to be monitored more closely when it comes to the presence of these autoantibodies during pregnancy. 

Signs and Symptoms 

There are several signs and symptoms associated with lupus, and they can vary in each patient. 

Patients dealing with lupus can develop rashes such as: 

  • An oval-shaped or a round rash that’s red (a discoid rash) 
  • A butterfly-shaped rash that spreads across the face (a malar rash) 
  • A rash spread across different body parts subject to sun exposure (i.e., face, arms, legs) 

Often in people with subacute cutaneous lupus and discoid lupus, a rash is the only symptom.  

Individuals with other types of lupus may present a combination of these signs or symptoms: 

  • Fever 
  • Muscle pain 
  • Continuing fatigue  
  • Chest pain 
  • Hair loss 
  • Fingers changing color following stress or exposure to cold (Raynaud phenomenon) 
  • Arthritis 
  • Swollen lymph nodes 
  • Anemia 
  • Joint pain like with arthritis but with no or little damage to the joint 
  • Sensitivity to sunlight 
  • Mouth ulcers 
  • Organ damage or inflammation (i.e., lungs, heart, central nervous system, kidneys, lining of the heart, and/or blood vessels) 

These symptoms can vary both in consistency and intensity, depending on the individual.  In some cases, a person may see an extreme bout of symptoms for a few months before seeing them taper off for a bit.  The return of these chronic symptoms can vary with flare-ups becoming an issue depending on specific triggers such as a person’s health, stress levels, external exposure (i.e., sunlight), pregnancies, or birth. 

A person that is dealing with lupus can also become prone to developing infections. These can include infections such as UTIs (or urinary tract infections), yeast infections, herpes, shingles, respiratory infections, and salmonella because their immune system isn’t running in top gear all the time. This can also lead to additional concerns about tissue death, miscarriages, and pre-eclampsia.  

Tests and Diagnosis 

The diagnostic process behind lupus can be difficult at the best of times due to the extensive list of symptoms. These symptoms also come and go making it difficult to come up with a comprehensive evaluation of the patient and their condition. Certain tests can be run to rule out other causes based on the symptoms that are present. 

The American College of Rheumatology took the time to develop classification criteria to assist physicians during the early-80s. These criteria were designed to assist with diagnosing SLE as it is the most common type of lupus; however, the criteria were updated against during the late-90s.  

These criteria include the following symptoms.  A person exhibiting at least 4 of the 11 following signs or symptoms may be diagnosed as having lupus:  

  • An oval-shaped or a round-shaped rash that is red (a discoid rash) 
  • A butterfly-shaped rash spread across the face (a malar rash) 
  • Mouth sores and/or nose ulcers 
  • Photosensitivity (consistent development of rashes on body parts exposed to sunlight (face, arms, legs, hands) 
  • Arthritis across multiple joints including tenderness, swelling, and/or accumulation of fluid in the joints; SLE-associated arthritis is non-erosive, so the bones near the affected joints are not damaged 
  • Kidney issues – elevated protein levels within the urine and/or cellular cysts 
  • Hematologic (blood) disorder – low white blood cell count, anemia, or low platelet count 
  • Pericarditis (inflammation of the lining around the heart) or pleuritis (inflammation of the lining around the lungs) 
  • Neurologic disorder – psychosis and/or seizures without relevant causes 
  • Positive blood test (antinuclear antibodies) – high levels can indicate SLE or drug-induced lupus (assuming specific medications were consumed) 
  • Positive blood test (anti-double-stranded DNA, cardiolipin antibody, anti-Sm (Smith) antibody, or lupus anticoagulant) or a false-positive test for syphilis (i.e., the person tests positive but doesn’t actually have the disease)  

It was in 2012 when the Systemic Lupus International Collaborating Clinics group took the time to verify the ACR’s SLE classification criteria.  The revision that was made stated that a person could be classified for having SLE if there was proven kidney-related involvement (lupus nephritis) after a complete biopsy.  This would also involve the presence of anti-dsDNA or ANA antibodies along with at least 4/11 diagnostic criteria, one or more symptoms, and a positive test. 

There are several lab tests available for spotting these autoantibodies in the system for a lupus diagnosis. 

These can include: 

Additional tests that can be used to diagnose a patient with lupus include: 

  • Complete Blood Count (CBC) – Helpful for spotting signs of low white blood cell count, low platelet count, and anemia as may occur in patients with lupus 
  • Urinalysis – Can show urinary casts, blood, or protein in the urine  
  • Serum protein electrophoresis (SPEP) – Helpful for spotting gamma globulin protein levels often cited alongside conditions such as SLE 
  • Comprehensive metabolic panel (CMP) – Ideal for assessing the patient’s liver and kidney production along with blood glucose levels, blood proteins, and acid/base balance. 
  • Erythrocyte sedimentation rate (ESR or SED rate) – Can help showcase signs of inflammation, which is commonly seen in patients with lupus and/or other relevant inflammatory conditions 
  • Complement 3 (C-3), CH50, and C4 – Can assist with understanding decreased production in the immune system but can also be an indicator of malaria, shock, or gram-negative septicemia  
  • C-reactive protein (CRP) – Can help spot inflammation  
  • Cryoglobulin – Can come back positive for high levels of this abnormal protein in the blood that will precipitate with reduced body temperature, leading to blockage of the blood vessels 

Non-laboratory tests include X-rays and other imaging used to look at particular organs potentially affected by lupus.  Also, a kidney biopsy may be run to find any changes that could point to lupus and/or guide treatment. 

 

What is Lupus (SLE)?

Lupus, also known by the formal name Systemic lupus erythematosus (SLE), is a chronic (long-term) autoimmune disorder where the body’s own immune system attacks healthy tissue. This causes inflammation, swelling, and pain in the body, most commonly in joints, skin, and organs. 

According to the National Resource Center on Lupus and the Lupus Foundation of America, more than 16,000 new cases of Lupus are confirmed each year in the United States, with an estimated 1.5 million Americans living with Lupus today. Many folks have heard about Lupus from celebrities that have the disease, such as singers Seal, Selena Gomez, and Paula Abdul. 

 

 

One of the methods used to confirm a Lupus diagnosis is to perform lab tests of the patient’s blood. These tests will screen for the presence of antibodies, specifically anti-nuclear antibodies (ANA), and other components of the blood to identify if the immune system is working abnormally. 

The cause and risk factors of Lupus are not well understood yet. There is no prevention or cure. Treatment efforts are focused on reducing the patient’s symptoms, especially pain and swelling. Lupus will require a lifetime of care, but celebrities suffering from the disease provide evidence and inspiration that individuals with Lupus can live successful lives.

What are the Risk Factors for Lupus (SLE)?

The following groups appear to be more likely to get Lupus:

  • 9 out of 10 Lupus patients are women, mostly aged 15-44
  • Certain racial or ethnic groups, including African American, Hispanic, and Pacific Islander
  • People who have a family member with Lupus

Due to the weakened immune system, Lupus patients are more susceptible to infections such as respiratory, yeast, urinary tract, herpes, and shingles. People suffering from repeat infections such as this should investigate if they may have an autoimmune disorder. 

Interestingly, Lupus patients may also have a false positive test for syphilis; meaning the blood test says a person has syphilis, but they actually do not. So individuals receiving a surprise and unexplainable diagnosis of syphilis should consider Lupus as a possibility.

What Causes Lupus (SLE)?

Doctors do not know what causes Lupus. Leading theories usually involve a complex mix of genes, hormones, and environmental factors. 

Theories about the cause of Lupus are primarily based on the risk factors for Lupus (the groups most likely to get the disease). 

  • With 9 out of 10 Lupus patients being female, it is assumed that hormones such as estrogen play a role. 
  • With Lupus appearing within families and races, it is assumed there is a genetic component; scientists have narrowed down a list of 50 genes that Lupus patients share, which may play a role.
  • But some people at high risk, such as an identical twin, never get the disease, so there is an assumed environmental component; something that triggers the disease to start. 

Lupus patients report certain environmental changes trigger their symptoms to flare-up, including increased exposure to sunlight, exercise, and sudden stress. Scientists are researching if these factors play a role in causing Lupus.

What are the Symptoms of Lupus (SLE)?

Lupus is a very challenging disease to recognize and diagnose because no two people have identical symptoms, and its symptoms look like many other diseases. In addition, Lupus symptoms are known to come and go in rounds of flares and temporary remissions. Sometimes the symptoms are overwhelming and sometimes they are barely noticeable. Symptoms can also change over time. It takes six years for the average Lupus patient to finally be diagnosed with the disease.

The most common symptoms of Lupus are:

  • Skin rashes; especially a butterfly-shaped rash on the cheeks and nose
  • Pain or swelling in the joints (arthritis)
  • Swelling in the feet and the eye area
  • Extreme fatigue
  • Sjogren's syndrome: dryness of mouth, eyes, and other body parts that self-lubricate
  • Low fevers
  • Headaches
  • Sensitivity to sunlight or fluorescent light
  • Chest pain when deeply breathing
  • Hair loss
  • Sores in the mouth and nose
  • Fingers and toes turning white/blue and feeling numb

According to the Lupus Foundation of America, 65% of people with Lupus say having chronic pain is the worst part of the disease. 76% of Lupus patients have to cut back on social activities due to extreme fatigue. And 89% of Lupus patients can no longer work full-time jobs due to complications from Lupus.

How is Lupus (SLE) Confirmed?

Lupus is a challenging disease to diagnose, and confirming the disease may involve a complicated review of symptoms and family history. However, there are blood tests that can help indicate that the body is suffering from a disease such as Lupus.

The most common blood tests for Lupus search for Anti-nuclear antibodies (ANA), which are found in almost all Lupus patients

  • ANA Pattern
  • ANA Screen, IFA
  • Anti-Nuclear Ab Titer

And searching for other antibodies that are common with autoimmune disorders

Other tests look at the blood’s ability to clot and look at red blood cells and white blood cells:

As well as the body's ability to attack infections:

There is no single test that can confirm Lupus, which is why testing for Lupus requires so many different blood tests. However, seeing any of the biomarkers out of range can certainly hint at Lupus. These test results can then be combined with physical symptoms and family history to make a Lupus diagnosis.

To simplify SLE testing and ensure that crucial biomarkers are analyzed, Ulta Lab Tests offers the Systemic Lupus Erythematosus (SLE) Comprehensive Diagnostic Panel that tests for the following 12 key biomarkers:
 
        1. ANA Pattern
        2. ANA Screen, IFA
        3. Anti-Nuclear Ab Titer
        4. Chromatin (Nucleosomal)
        5. Complement Component C3c
        6. Complement Component C4c
        7. Complement, Total (Ch50)
        8. Dna (Ds) Antibody
        9. Rnp Antibody
        10. Sjogren's Antibody (Ss-A)
        11. Sjogren's Antibody (Ss-B)
        12. Sm Antibody

What Happens if Lupus (SLE) Goes Untreated?

If left untreated, Lupus patients can expect inflammation to continue to worsen until major organs are impacted. The most serious consequences of Lupus are:

  • Inflammation of the kidneys, eventually leading to kidney failure and/or the need for dialysis and/or kidney transplant.
  • Inflammation of the nervous system causing memory problems, headaches, and strokes
  • Inflammation of blood vessels in the brain causing fevers, seizures, and changes to behavior
  • Inflammation of the heart and arteries leading to heart attack
  • Inflammation of the skin causing rashes, ulcers, and sores 

The damage from Lupus can accrue over time. Those suffering from severe symptoms at the start of the disease, or suffering severe symptoms for more than two years, or have four or more severe episodes of the disease, are more likely to suffer organ damage.

Research is also showing that even brief periods where symptoms are reduced can be enough to reduce the accrued damage to organs. This demonstrates the importance of early diagnosis and treating Lupus effectively.

How is a Lupus (SLE) Treated?

There is currently no way to prevent or cure Lupus (SLE). Treatment efforts are focused on relieving symptoms, controlling the immune system, and slowing down the worsening of inflammation. Unfortunately, due to the wide variety of symptoms of Lupus, and the fact symptoms can change over time, a Lupus patient may spend many years trying to find the right combination of treatments to relieve their symptoms.

Examples of medications commonly used to treat Lupus symptoms include:

  • Anti-inflammatories to help with pain and swelling
  • Antimalarials to control skin rashes
  • Anticoagulants for blood clots
  • Immunosuppressives and biologics to help control the immune system
  • Steroids to help inflammation

Lupus patients should also minimize the environmental factors that trigger their symptoms to flare, such as exposure to sunlight, exercise, and sudden stresses.

Benefits of Using Ulta Lab Tests for Lupus (SLE) Testing

One of the primary methods of diagnosing SLE Lupus is through blood tests that detect unusual antibodies in the blood, especially antinuclear antibodies (ANA). Because Lupus is so difficult to diagnose, multiple blood tests may be needed to look at a wide range of biomarkers. Ulta Lab Tests has individual and panels of SLE blood tests available for purchase today. 

Ulta Lab Tests provide comprehensive and affordable laboratory testing and health screening services directly to consumers. Our mission is to enable individual consumers across America to take control of their healthcare by having direct access to the quality lab tests of their choice. Consumers are now able to be proactive in the early detection and prevention of disease and do so with complete control of their healthcare costs.

Our services are perfect for a wide variety of people, such as:

  • Anyone looking to proactively monitor their health
  • Those with high deductible insurance plans
  • People looking to use HSA/FSA funds
  • The uninsured and self-employed
  • Businesses looking to frequently test employees

We offer tests that are highly accurate and reliable so you can make informed decisions about your health. 

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Order your Lupus lab tests today and your test results will be provided to you securely and confidentially online in 24 to 48 hours for most tests.

Take charge of your health with Ulta Lab Tests.